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Curcumin, Curcumin98, Curcumin x4000

Turmeric has long been revered as the foundation of an herbal program for health. In India's system of Ayurvedic medicine, it has been recognized for thousands of years as a key balancing and detoxifying herb and is considered to be one of the very best all-around herbs for general well-being. Curcumin, is the main biologically active part of Turmeric, which only contains 4% curcumin; whereas Curcumin98 contains 98%. Over 500 references to articles on turmeric and curcumin have been published in peer reviewed professional journals.

Curcumin, the active ingredient in the herb turmeric, is being investigated for use in Alzheimer's disease due to its potent anti-inflammatory action (Joe 1997; Grilli 1999)

If that is not enough, Turmeric has been used for thousands of years by Indian Women to make their skin beautiful and blemish free.

No side effects have been found taking high doses of Curcumin; rare cases of stomach upset or diarrhoea may be resolved by temporarily, reducing the dosage and taking with food.


Do not use Curcumin if you suffer from gallstones or a blocked bile duct, as it increases the production of bile acid.
If you are taking Warfarin or other blood thinning medication, consult your doctor before taking Curcumin.
Curcumin Treating eye disorders

Curcumin is apparently more than your typical kitchen spice. It's the substance that gives ginger its yellowish colour, and it has been implicated in the treatment of certain eye diseases and conditions. One of those is known as chronic anterior uveitis (CAU), an inflammatory condition of the vascular layer of the eye, particularly the area comprising the iris. In one small study, Curcumin was given orally to 32 chronic anterior uveitis patients who were divided into two groups. The first group received Curcumin alone, whereas the second group received a combination of Curcumin and antitubercular treatment. Amazingly, all of the patients treated with Curcumin alone improved, compared to a response rate of 86% among those receiving the combination therapy. The researchers concluded that Curcumin was just as effective as corticosteroid therapy, the only available standard treatment for chronic anterior uveitis at present, adding that "the lack of side effects with Curcumin is its greatest advantage compared with corticosteroids."

Similar research using rats and rabbits found that Curcumin effectively inhibited chemically induced cataract formation, even at very low dietary levels. The same study also found, for the first time, that this type of induced cataract may be accompanied by apoptosis of epithelial cells in the eye and that Curcumin may lessen the apoptotic effect. In one of the earliest studies examining Curcumin as a potential cataract therapy, researchers fed two groups of rats, diets that included corn oil, or a combination of Curcumin and corn oil for 14 days. Afterward, their lenses were removed and examined for the presence of lipid peroxidation. The scientists discovered that "the lenses from Curcumin-treated rats were much more resistant to. induced opacification than were lenses from control animals."

Awasthi S et al. Curcumin protects against 4-hydroxy-2-trans-nonenal-induced cataract formation in rat lenses. Am J Clin Nutr 1996 Nov;64 (5):761-6.

Lal B et al. Efficacy of curcumin in the management of chronic anterior uveitis. Phytother Res 1999 Jun;13(4):318-22.

Pandya U et al. Dietary curcumin prevents ocular toxicity of naphthalene in rats. Toxicol Lett 2000 Jun 5;115(3):195-204.

Curcumin may block the progression of Multiple Sclerosis (MS)
NEW ORLEANS (Reuters Health) - Preliminary studies in rats suggest that curcumin, a compound found in the curry spice turmeric, may block the progression of multiple sclerosis (MS).

According to researcher Dr. Chandramohan Natarajan of Vanderbilt University in Nashville, Tennessee, rats with an MS-like illness showed little or no signs of disease symptoms after being injected with curcumin, while animals without the treatment went on to severe paralysis.

"We got a very good inhibition of the disease by treating with curcumin," Natarajan told Reuters Health. He presented the findings here Tuesday at the annual Experimental Biology 2002 conference.

No one knows what causes multiple sclerosis, in which the body's immune system attacks the protective myelin sheath surrounding nerve fibers in the brain and spine. Symptoms of multiple sclerosis include muscle weakness and stiffness, balance and coordination problems, numbness and vision disturbances.

Interest in the potential neuroprotective properties of curcumin rose after studies found very low levels of neurological diseases such as Alzheimer's in elderly Indian populations. Added to this were studies confirming curcumin as a potent anti-inflammatory agent, effective in wound healing. And just last fall, researchers at the University of California, Los Angeles reported that curcumin appeared to slow the progression of Alzheimer's in mice.

In their 30-day study, Natarajan and co-researcher Dr. John Bright gave injections of 50- and 100-microgram doses of curcumin, three times per week, to a group of mice bred to develop a disease called experimental autoimmune encephalomyelitis (EAE)--an autoimmune condition used by researchers as a model for multiple sclerosis because it also results in the slow erosion of myelin. They then watched the rats for signs of MS-like neurological impairment.

By day 15, rats who had not received curcumin developed EAE to such an extent that they displayed complete paralysis of both hind limbs, according to Natarajan.

In contrast, rats given the 50-microgram dose of the curry compound showed only minor symptoms, such as a temporarily stiff tail. And rats given the 100-microgram dose appeared completely unimpaired throughout the 30 days of the study.

The results didn't really surprise Natarajan. "In Asian countries, such as India, China, who are eating more spicy foods, more yellow compounds like curcumin...there are only very, very rare reports of MS," he pointed out. He said the doses the rats received were roughly equivalent in human terms to those found in a typical Indian diet.

Just how curcumin might work to thwart the progression of demyelinization remains unclear. But the Nashville researchers believe it may interrupt the production of IL-12, a protein that plays a key role in signaling immune cells to launch their assault on the myelin sheath.

Natarajan stressed that "we have to do a lot of work on this," including examining other potential mechanisms by which curcumin slows EAE and, potentially, MS.

The work remains preliminary, and MS patients should follow their doctor's advice when it comes to treating the disease. Still, Natarajan said adding a little curry to the diet couldn't hurt. "I think using this spice in their food could be of help," he said

Curcumin has potent anti-amyloidogenic effects for Alzheimer's beta-amyloid fibrils in vitro.

Ono K, Hasegawa K, Naiki H, Yamada M. J Neurosci Res. 2004 Mar 15;75(6):742-50.

Kanazawa University Graduate School of Medical Science, Kanazawa, Japan.
Inhibition of the accumulation of amyloid beta-peptide (Abeta) and the formation of beta-amyloid fibrils (fAbeta) from Abeta, as well as the destabilization of preformed fAbeta in the central nervous system, would be attractive therapeutic targets for the treatment of Alzheimer's disease (AD). We reported previously that nordihydroguaiaretic acid (NDGA) and wine-related polyphenols inhibit fAbeta formation from Abeta(1-40) and Abeta(1-42) and destabilize preformed fAbeta(1-40) and fAbeta(1-42) dose-dependently in vitro. Using fluorescence spectroscopic analysis with thioflavin T and electron microscopic studies, we examined the effects of curcumin (Cur) and rosmarinic acid (RA) on the formation, extension, and destabilization of fAbeta(1-40) and fAbeta(1-42) at pH 7.5 at 37 degrees C in vitro. We next compared the anti-amyloidogenic activities of Cur and RA with NDGA. Cur and RA dose-dependently inhibited fAbeta formation from Abeta(1-40) and Abeta(1-42), as well as their extension. In addition, they dose-dependently destabilized preformed fAbetas. The overall activities of Curcumin, RA, and NDGA were similar. The effective concentrations (EC(50)) of Curcumin, RA, and NDGA for the formation, extension, and destabilization of fAbetas were in the order of 0.1-1 microM. Although the mechanism by which Curcumin and RA inhibit fAbeta formation from Abeta and destabilize preformed fAbeta in vitro remains unclear, they could be a key molecule for the development of therapeutics for AD.
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All round Herbs for general well-being. Curcumin, is the main biologically active part of Turmeric. Over 500 references to articles on Turmeric and Curcumin have been published in peer reviewed professional journals.

It has been identified in pharmacology as: antibacterial, antiviral, anti fungal, anti yeast, antiallergenic,
anti- inflammatory, anti- oxidant, anti- spasmodic, carminative, diuretic, and anti-tumour

Turmeric and Curcumin have traditionally been used to support those suffering from pain and inflammation: acne, allergies, ascites, auto-immune disorders, burns, chicken pox, diabetes, digestive disorders, gallbladder problems, liver damage, liver disorders, skin rashes, tumours, ulcers and eye problems such as cataracts.

If that is not enough, Turmeric has been used for thousands of years by Indian Women to make their skin beautiful and blemish free.

No side effects have been found taking high doses of Curcumin; rare cases of stomach upset or diarrhoea may be resolved by temporally, reducing the dosage and taking with food.

There are plenty of New Studies which show benefits with: Alzheimer's, Kidney-Disease, Liver Disease, Anti-Carcinogenic and Anti-Cancer, Anti-Arthritic, Anti-Coagulant, Lowering-LDL-and-Raising HDL-Cholesterols, Anti-Viral

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